Before we delve into homeopathic treatment of Bell’s palsy let us discuss its allopathic or conventional treatment available.
- Chances of recovery are maximum if treatment is started within three days of appearance of symptoms.
- Corticosteroids (e.g. Prednisolone) in acute phase of Bell’s palsy having anti-inflammatory action on nerve.
- Anti-viral agents in cases of infections with herpes simplex virus.
Now let us discuss role of homeopathy.
- Homeopathy has documented remedies for Bell’s palsy which have been found very effective.
- Muscle tone of the facial muscles improve.
- Inability to close the eye, hyperacusis (acute hearing) and other symptoms can be effectively taken care of.
- Prolonged homeopathic treatment would improve the residual symptoms of Bell’s palsy.
- Steroids used during allopathic treatment usually have side effects which can be very well managed with concurrent homeopathic remedies.
- Some of the remedies used in Bell’s palsy are Agaricus, Cadmium sulph, Cocculus, Baryta carb, Graphites, Kali phos, Pulsatilla, Silecea, Tellurium (Note: Take your medications exactly as prescribed by your doctor, including at the right times and for the full length of your prescribed treatment).
There are 20 medicines which give great relief in Bell’s palsy or facial nerve palsy. However, the correct choice and the resulting relief is a matter of experience and right judgment on the part of the doctor. The treatment is decided after thorough case taking of the patient. Thus, homeopathic remedies are tailor made unlike allopathy in which all patients receive the same drugs although trade name may be different.
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What is Bell’s Palsy
- Occurs due to sudden paralysis or compression of facial nerve (VII cranial nerve) in the stylomastoid foramen.
- It is a type of lower motor lesion of stylomastoid foramen.
- Underlying cause is unknown.
- Immediate cause may include neuritis and edema.
- Onset may be sudden or may develop over several hours.
Symptoms / Effects
- It causes paralysis of the ipsilateral half of the face, i.e. both upper and lower quadrant of the same side of the lesion.
- Distortion of features due to muscle tone on unaffected side.
- Asymmetry of corner of mouth.
- Inability to close the eye on the same side.
- Disappearance of nasolabial fold.
- Loss of wrinkling of skin on forehead of the same side.
- Drooping and loss of the facial expressions on the same side.
Additional features (may be present)
- Loss of lachrymation.
- Loss of stapedial reflex.
- Loss of taste from anterior 2/3 of tongue.
- Lack of salivation.
- Purely clinical (mostly).
- Trigeminal blink reflex – to study various post paralysis sequale.
- Gadolinium contrast MRI: for enhancement of internal acoustic meatal segment on the affected side (nonspecific; should not be done routinely).
Poor prognostic factors
- Old age
- Diabetes mellitus
- Complete facial weakness
(One third of patients may have incomplete recovery)
- Hemifacial spasm
- Synkinesis (involuntary twitching of muscles along with voluntary action)
- Sweating while eating or physical exertion
- Ipsilateral crocodile tears and jaw winking
- Electrotherapy massage